##Vega-Lugo JA, Parodi-Rullán RM, Soto-Prado J, Chapa-Dubocq X, Javadov S (2017). The effects of myocardial infarction with or without subsequent reperfusion on respiratory function of mitochondria in rat heart. The FASEB Journal.;31(1 Supplement):1080.8-8.##Wu Y, Yin X, Wijaya C, Huang M-H, McConnell BK (2011). Acute myocardial infarction in rats. Journal of visualized experiments: JoVE. (48).##Lujan HL, DiCarlo SE (2013). Mimicking the endogenous current of injury improves post-infarct cardiac remodeling. Medical hypotheses.;81(4):521-3.##Zornoff LA, Paiva SA, Minicucci MF, Spadaro J (2009). Experimental myocardium infarction in rats: analysis of the model. Arquivos brasileiros de cardiologia.;93(4):434-40.##Cohn JN, Ferrari R, Sharpe N (2000). Cardiac remodeling—concepts and clinical implications: a consensus paper from an international forum on cardiac remodeling. Journal of the American College of Cardiology.;35(3):569-82.##Heather LC, Carr CA, Stuckey DJ, Pope S, Morten KJ, Carter EE, et al (2009). Critical role of complex III in the early metabolic changes following myocardial infarction. Cardiovascular research.;85(1):127-36.##Sena S, Hu P, Zhang D, Wang X, Wayment B, Olsen C, et al (2009). Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarction. Journal of molecular and cellular cardiology.;46(6):910-8.##Tsutsui H, Kinugawa S, Matsushima S (2008). Mitochondrial oxidative stress and dysfunction in myocardial remodelling. Cardiovascular research.;81(3):449-56.##Rosca MG, Vazquez EJ, Kerner J, Parland W, Chandler MP, Stanley W, et al (2008). Cardiac mitochondria in heart failure: decrease in respirasomes and oxidative phosphorylation. Cardiovascular research.;80(1):30-9.##Hausenloy DJ, Yellon DM (2003). The mitochondrial permeability transition pore: its fundamental role in mediating cell death during ischaemia and reperfusion. Academic Press.##Halestrap AP (2009). Mitochondria and reperfusion injury of the heart—a holey death but not beyond salvation. Journal of bioenergetics and biomembranes.;41(2):113-21.##Zhou S, Sun W, Zhang Z, Zheng Y (2014). The role of Nrf2-mediated pathway in cardiac remodeling and heart failure. Oxidative medicine and cellular longevity.##Hori M, Nishida K (2008). Oxidative stress and left ventricular remodelling after myocardial infarction. Cardiovascular research.81(3):457-64.##Huss JM, Kelly DP (2005). Mitochondrial energy metabolism in heart failure: a question of balance. Journal of Clinical Investigation.;115(3):547.##Finck BN, Kelly DP (2007). Peroxisome proliferator–activated receptor γ coactivator-1 (PGC-1) regulatory cascade in cardiac physiology and disease. Circulation.115(19):2540-8.##Chan DC (2012). Fusion and fission: interlinked processes critical for mitochondrial health. Annual review of genetics.46:265-87.##Hall A, Burke N, Dongworth R, Hausenloy D (2014). Mitochondrial fusion and fission proteins: novel therapeutic targets for combating cardiovascular disease. British journal of pharmacology. 171(8):1890-906.##Iglewski M, Hill JA, Lavandero S, Rothermel BA (2010). Mitochondrial fission and autophagy in the normal and diseased heart. Current hypertension reports.12(6):418-25.##Ong S-B, Hausenloy DJ (2010). Mitochondrial morphology and cardiovascular disease. Cardiovascular research.88(1):16-29.##Disatnik M-H, Ferreira JC, Campos JC, Gomes KS, Dourado PM, Qi X, et al (2013). Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction. Journal of the American Heart Association.2(5):e000461.## Jiang H-K, Wang Y-H, Sun L, He X, Zhao M, Feng Z-H, et al (2014). Aerobic interval training attenuates mitochondrial dysfunction in rats post-myocardial infarction: roles of mitochondrial network dynamics. International journal of molecular sciences.15(4):5304-22.##Icks A, Claessen H, Kirchberger I, Heier M, Peters A, Trentinaglia I, et al (2014). Mortality after first myocardial infarction in diabetic and non-diabetic people between 1985 and 2009. The MONICA/KORA registry. European journal of epidemiology.29(12):899-909.##Hosseini SH, Ghaemian A, Mehdizadeh E, Ashraf H (2014). Levels of anxiety and depression as predictors of mortality following myocardial infarction: A 5-year follow-up. Cardiology journal.21(4):370-7.##Ellison GM, Waring CD, Vicinanza C, Torella D (2012). Physiological cardiac remodelling in response to endurance exercise training: cellular and molecular mechanisms. Heart.98(1):5-10.##Weiner RB, Baggish AL (2012). Exercise-induced cardiac remodeling. Progress in cardiovascular diseases.54(5):380-6.##Lee Y, Min K, Talbert EE, Kavazis AN, Smuder AJ, Willis WT, et al (2012). Exercise protects cardiac mitochondria against ischemia–reperfusion injury. Medicine & Science in Sports & Exercise.44(3):397-405.##Quindry JC, Miller L, McGinnis G, Kliszczewicz B, Irwin JM, Landram M, et al (2012). Ischemia reperfusion injury, K ATP channels, and exercise-induced cardioprotection against apoptosis. Journal of Applied Physiology.113(3):498-506.##Yan Z, Lira VA, Greene NP (2012). Exercise training-induced regulation of mitochondrial quality. Exercise and sport sciences reviews.40(3):159.##Cartoni R, Léger B, Hock MB, Praz M, Crettenand A, Pich S, et al (2005). Mitofusins 1/2 and ERRα expression are increased in human skeletal muscle after physical exercise. The Journal of physiology.567(1):349-58.##Perry CG, Lally J, Holloway GP, Heigenhauser GJ, Bonen A, Spriet LL (2010). Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle. The Journal of physiology.;588(23):4795-810.##Kuzmicic J, del Campo A, López-Crisosto C, Morales PE, Pennanen C, Bravo-Sagua R, et al (2011). Mitochondrial dynamics: a potential new therapeutic target for heart failure. Revista Española de Cardiología (English Edition).64(10):916-23.##Finck BN, Kelly DP (2006). PGC-1 coactivators: inducible regulators of energy metabolism in health and disease. Journal of Clinical Investigation.116(3):615.##Huh JY, Mougios V, Kabasakalis A, Fatouros I, Siopi A, Douroudos II, et al (2014). Exercise-induced irisin secretion is independent of age or fitness level and increased irisin may directly modulate muscle metabolism through AMPK activation. The Journal of Clinical Endocrinology & Metabolism.99(11):E2154-E61.##Fukuda S, Kaga S, Sasaki H, Zhan L, Zhu L, Otani H, et al (2004). Angiogenic signal triggered by ischemic stress induces myocardial repair in rat during chronic infarction. Journal of molecular and cellular cardiology.36(4):547-59.##Samsamshariat SA, Samsamshariat ZA, Movahed M-R (2005). A novel method for safe and accurate left anterior descending coronary artery ligation for research in rats. Cardiovascular Revascularization Medicine.6(3):121-3.##Scherrer-Crosbie M, Thibault HB (2008). Echocardiography in translational research: of mice and men. Journal of the American Society of Echocardiography.21(10):1083-92.##Waring CD, Vicinanza C, Papalamprou A, Smith AJ, Purushothaman S, Goldspink DF, et al (2012). The adult heart responds to increased workload with physiologic hypertrophy, cardiac stem cell activation, and new myocyte formation. European heart journal.35(39):2722-31.##Kemi OJ, Haram PM, Loennechen JP, Osnes J-B, Skomedal T, Wisløff U, et al (2005). Moderate vs. high exercise intensity: differential effects on aerobic fitness, cardiomyocyte contractility, and endothelial function. Cardiovascular research.67(1):161-72.##Kraljevic J, Marinovic J, Pravdic D, Zubin P, Dujic Z, Wisloff U, et al (2013). Aerobic interval training attenuates remodelling and mitochondrial dysfunction in the post-infarction failing rat heart. Cardiovascular research.99(1):55-64.##Ong S-B, Subrayan S, Lim SY, Yellon DM, Davidson SM, Hausenloy DJ (2010). Inhibiting mitochondrial fission protects the heart against ischemia/reperfusion injury. Circulation.121(18):2012-22.##Din S, Mason M, Völkers M, Johnson B, Cottage CT, Wang Z, et al (2013). Pim-1 preserves mitochondrial morphology by inhibiting dynamin-related protein 1 translocation. Proceedings of the National Academy of Sciences.110(15):5969-74.##Leistner DM, Zeiher AM (2012). Novel avenues for cell therapy in acute myocardial infarction. Am Heart Assoc.##Yu T, Robotham JL, Yoon Y (2006). Increased production of reactive oxygen species in hyperglycemic conditions requires dynamic change of mitochondrial morphology. Proceedings of the National Academy of Sciences of the United States of America.103(8):2653-8.##Gomez-Lazaro M, Bonekamp NA, Galindo MF, Jordán J, Schrader M (2008). 6-Hydroxydopamine (6-OHDA) induces Drp1-dependent mitochondrial fragmentation in SH-SY5Y cells. Free Radical Biology and Medicine.44(11):1960-9.##Parker L, McGuckin TA, Leicht AS (2014). Influence of exercise intensity on systemic oxidative stress and antioxidant capacity. Clinical physiology and functional imaging.34(5):377-83.##St-Pierre J, Drori S, Uldry M, Silvaggi JM, Rhee J, Jäger S, et al (2006). Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators. Cell;127(2):397-408.##Liesa M, Borda-d'Água B, Medina-Gómez G, Lelliott CJ, Paz JC, Rojo M, et al (2008). Mitochondrial fusion is increased by the nuclear coactivator PGC-1β. PloS one.3(10):e3613.##Ding H, Jiang N, Liu H, Liu X, Liu D, Zhao F, et al (2010). Response of mitochondrial fusion and fission protein gene expression to exercise in rat skeletal muscle. Biochimica et Biophysica Acta (BBA)-General Subjects.1800(3):250-6.##Kang C, O'moore KM, Dickman JR, Ji LL (2009). Exercise activation of muscle peroxisome proliferator-activated receptor-γ coactivator-1α signaling is redox sensitive. Free Radical Biology and Medicine.47(10):1394-400.##