##
Verhees KJ,
Schols AM,
Kelders MC,
Op den Kamp CM,
van der Velden JL,
Langen RC. 2011. Glycogen synthase kinase-3β is required for the induction of skeletal muscle atrophy.
Am J Physiol Cell Physiol. 301: 995-1007.##
Fanzani A,
Conraads VM,
Penna F,
Martinet W. 2012. Molecular and cellular mechanisms of skeletal muscle atrophy: an update. Journal of cachexia, sarcopenia and muscle.
J Cachexia Sarcopenia Muscle. 3(3): 163-179.## Jespersen, J., Nedergaard, A., Andersen, L., Schjerling, P. & Andersen, J. 2011. Myostatin Expression during Human Muscle Hypertrophy and Subsequent Atrophy: Increased Myostatin with Detraining
Scand J Med Sci Sports. ;21(2):215-23.##
Paul PK1,
Gupta SK,
Bhatnagar S,
Panguluri SK,
Darnay BG,
Choi Y, et al. 2010. Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice. J Cell Biol. 191(7):1395-411.##Winkles, J.A. 2008. The TWEAK–Fn14 cytokine–receptor axis: discovery, biology and therapeutic targeting. Nat Rev Drug Discov. 7(5): 411-425.##Kumar, A., S. Bhatnagar, and P.K. Paul. 2012. TWEAK and TRAF6 regulate skeletal muscle atrophy.
Curr Opin Clin Nutr Metab Care. 15(3):233-9 .##
Dogra C,
Changotra H,
Mohan S,
Kumar A. 2006. Tumor necrosis factor-like weak inducer of apoptosis inhibits skeletal myogenesis through sustained activation of nuclear factor-κB and degradation of MyoD protein. J Biol Chem. 281(15):10327-10336.##
Dogra C,
Hall SL,
Wedhas N,
Linkhart TA,
Kumar A. 2007.Fibroblast growth factor inducible 14 (Fn14)is required forthe expression of myogenicregulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAKindependent functions of Fn14 during myogenesis.
J Biol Chem.. 282(20):15000-10.##
Clarke BA,
Drujan D,
Willis MS,
Murphy LO,
Corpina RA,
Burova E, et al. 2007. The E3 Ligase MuRF1 degrades myosin heavy chain protein in dexamethasone-treated skeletal muscle.
Cell Metab. 6(5):376-85.##
Bhatnagar S1,
Mittal A,
Gupta SK,
Kumar A. 2012. TWEAK causes myotube atrophy through coordinated activation of ubiquitin‐proteasome system, autophagy, and caspases.
J Cell Physiol. 227(3):1042-51.##
Peterson JM1,
Bakkar N,
Guttridge DC. 2011. NF-kB Signaling in Skeletal Muscle Health and Disease.
Curr Top Dev Biol. 96:85-119.##
Mittal A1,
Bhatnagar S,
Kumar A,
Lach-Trifilieff E,
Wauters S,
Li H, et al. 2010. The TWEAK–Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice. J Cell Biol. 188(6):833-49.##Wu, C.-L., S.C. Kandarian, and R.W. Jackman. 2011. Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3. PloS one. 6(1):e16171.##
Hindi SM1,
Mishra V,
Bhatnagar S,
Tajrishi MM,
Ogura Y,
Yan Z, et al. 2014. Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program. FASEB J. 28(3):1398-1411.##
Tajrishi MM,
Sato S,
Shin J,
Zheng TS,
Burkly LC,
Kumar A.2014. The TWEAK–Fn14 dyad is involved in age-associated pathological changes in skeletal muscle.
Biochem Biophys Res Commun. 446(4):1219-1224.##Meng, S.-J. and L.-J. Yu. 2010. Oxidative stress, molecular inflammation and sarcopenia.
Int J Mol Sci. 11(4):1509-26.##
Sheffield-Moore M,
Yeckel CW,
Volpi E,
Wolf SE,
Morio B,
Chinkes DL, et al. 2004. Postexercise protein metabolism in older and younger men following moderate-intensity aerobic exercise.
Am J Physiol Endocrinol Metab. 287(3): E513-E522.##
Raue U,
Slivka D,
Jemiolo B,
Hollon C,
Trappe S. 2006. Myogenic gene expression at rest and after a bout of resistance exercise in young (18–30 yr) and old (80–89 yr) women.
J Appl Physiol . 101(1):53-59.##
Cunha TF,
Bacurau AV,
Moreira JB,
Paixão NA,
Campos JC,
Ferreira JC,et al. 2012. Exercise training prevents oxidative stress and ubiquitin-proteasome system overactivity and reverse skeletal muscle atrophy in heart failure. PLoS One. 7(8):e41701.##
Shefer G,
Rauner G,
Yablonka-Reuveni Z,
Benayahu D. 2010. Reduced satellite cell numbers and myogenic capacity in aging can be alleviated by endurance exercise. PLoS One. 5(10): e13307-e13307.##
Gibala MJ1,
Little JP,
van Essen M,
Wilkin GP,
Burgomaster KA, et al. 2006 Short‐term sprint interval versus traditional endurance training: similar initial adaptations in human skeletal muscle and exercise performance.
J Physiol. 575 (Pt 3):901-11.##Snijders, T., L.B. Verdijk, and L.J. van Loon. 2009. The impact of sarcopenia and exercise training on skeletal muscle satellite cells. Ageing Res Rev. 8(4):328-338.##
Ferreira JC1,
Bacurau AV,
Bueno CR Jr,
Cunha TC,
Tanaka LY,
Jardim MA, et al. 2010. Aerobic exercise training improves Ca2+ handling and redox status of skeletal muscle in mice.
Exp Biol Med (Maywood) . 235(4): 497-505.##Kim SH, Chung JM. 1992. An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat. Pain. 50(3): 355-63.##Hafstad AD, Boardman NT, Lund J, Hagve M, Khalid AM, Wisloff U, et al. 2011. High intensity interval training alters substrate utilization and reduces oxygen consumption in the heart. J Appl Physiol. 111(5):1235-41.##Tal M, Bennett GJ. 1994. Extra–territorial pain in rat with a peripheral mononeuropathy: mechano–hyperalgesia and mechano – allodynia in the territory of an uninjerd nerve. Pain. 57: 375-382.##Nilwik, R., Snijders, T., Leenders, M., Groen, B. B., Van Kranenburg, J., Verdijk, L. B. & Van Loon, L. J. 2013. The Decline In Skeletal Muscle Mass With Aging Is Mainly Attributed To A Reduction In Type Ii Muscle Fiber Size. Experimental Gerontology. 48, 492-498.##Muller, F. L., Song, W., Jang, Y. C., Liu, Y., Sabia, M., Richardson, A. & Van Remmen, H. Denervation-Induced Skeletal Muscle Atrophy Is Associated With Increased Mitochondrial Ros Production. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2007; 293, R1159-R1168.##Rowan, S. L., Rygiel, K., Purves-Smith, F. M., Solbak, N. M., Turnbull, D. M. & Hepple, R. T. 2012. Denervation Causes Fiber Atrophy and Myosin Heavy Chain Co-Expression in Senescent Skeletal Muscle. Plos One; 7, E29082.##Verdijk, L.B., et al. 2007. Satellite cell content is specifically reduced in type II skeletal muscle fibers in the elderlyAmerican Journal of Physiology-Endocrinology and Metabolism; 292(1): p. E151-E157.##Bonaldo, P. & Sandri, M. Cellular and Molecular Mechanisms of Muscle Atrophy. Disease Models & Mechanisms, 2013; 6, 25-39.##Sandri, M., Barberi, L., Bijlsma, A., Blaauw, B., Dyar, K., Milan, G., Mammucari, C. 2013. Meskers, C., Pallafacchina, G. & Paoli, A. Signalling Pathways Regulating Muscle Mass in Ageing Skeletal Muscle. The Role of the Igf1-Akt-Mtor-Foxo Pathway. Biogerontology. 14:303-323.##Tajrishi, M.M., Sato, S., Shin, J., Zheng, T.S., Burkly, L.C., Kumar, A. 2014. The TWEAK–Fn14 dyad is involved in age-associated pathological changes in skeletal muscle. Biochemival and Biophysical Research Communication.446(4): 1219-1224.##
Mittal, A.,
Bhatnagar, S.,
Kumar, A.,
Lach-Trifilieff, E.,
Wauters, S.,
Li, H.,
Makonchuk, D.Y.,
Glass, D.J.,
Kumar, A. 2010. The TWEAK–Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice. Journal of Cellular Biology. 188(6): 833-49.##
Shi, J.,
Jiang, B.,
Qiu, Y.,
Guan, J.,
Jain, M.,
Cao, X.,
Bauer, M.,
Su, L.,
Burkly , L.C.,
Leone, T.C.,
Kelly, D.P.,
Liao, R. 2013. PGC1alpha plays a critical role in TWEAK-induced cardiac dysfunction. PLoS One. 8(1): e54054.##Kazemi A. 2018. The effect of the mechanical unloading of lower limb on MST1 and Atrogin1 gene expression in Plantaris and soleus muscles of Wistar rats. Journal of Knowledge & Health 13(3):33-41.##Gomes, AV.,
Waddell, DS.,
Siu, R.,
Stein, M.,
Dewey, S.,
Furlow, J.D.,
Bodine, S.C. 2012. Upregulation of proteasome activity in muscle RING finger 1-null mice following denervation. FASEB Journal. 26(3): 2886-99.##Arbat-Plana, A., Cobianchi, S., Herrando-Grabulosa, M., Navarro, X., Udina, E. 2017. Endogenous modulation of TrkB signaling by treadmill exercise after peripheral nerve injury. Neuroscience. 340: 188-200.##English, A.W., Wilhelm, J.C., Sabatier, M.J. 2011. Enhancing recovery from peripheral nerve injury using treadmill training. Annals of Anatomy.193(4): 354-61.##Gleeson, M., Bishop, N. C., Stensel, D. J., Lindley, M. R., Mastana, S. S. & Nimmo, M. A. 2011. The Anti-Inflammatory Effects Of Exercise: Mechanisms and implications for the prevention and treatment of disease. Nature Reviews Immunology. 11(9): 607-615.##Moreira, J.B., Bechara, L.R., Bozi, L.H., Jannig, P.R., Monteiro, A.W., Dourado, P.M., Wisløff, U., Brum, P.C. 2013. High- versus moderate-intensity aerobic exercise training effects on skeletal muscle of infarcted rats. Journal of Applied Physiology. 114(8): 1029-41.##Little, J.P., Safdar, A., Bishop, D., Tarnopolsky, MA., Gibala, M.J. 2011. An acute bout of high-intensity interval training increases the nuclear abundance of PGC-1α and activates mitochondrial biogenesis in human skeletal muscle. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 300(6): R1303-10.##Gurd, B.J., Perry, C.G., Heigenhauser, G.J., Spriet, L.L., Bonen, A. 2010. High-intensity interval training increases SIRT1 activity in human skeletal muscle. Applied Physiology, Nutrition and Metabolism. 35(3): 350-7.##
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