##Rosa, E. & Fahnestock, M. 2014. Amyloid-Beta, BDNF, and the Mechanism of Neurodegeneration in Alzheimer’s Disease. Handbook of Neurotoxicity. Springer.##Deane, R., Sagare, A., Hamm, K., Parisi, M., et al. 2008. ApoE isoform-specific disruption of amyloid β peptide clearance from mouse brain. J. Clin. Invest. 118: 4002–4013.##Sagare A, Deane R, Bell RD, et al.2007. Clearance of amyloid-b by circulating lipoprotein receptors. Nat Med; 13:1029–31.##Wrann CD., White, JP., Salogiannis J, Laznik-Bogoslavski D, Wu J, Ma D, Lin JD, Greenberg ME. & Spiegelman BM. 2013. Exercise induces hippocampal BDNF through a PGC-1α/FNDC5 pathway. Cell metabolism, 18, 649-659.##Adlard, A., Perreau, V.M., Pop, V., Cotman, C.W. 2005. Voluntary exercise decreases amyloid load in a transgenic model of Alzheimer’s disease. J Neurosci. 25(17):4217-4221.##Yuede, C.M., Zimmerman, S.D., Dong, H., Kling, M.J., Bero, A.W., Holtzman, D.M., Timson, B.F., Csernansky, J.G., 2009. Effects of voluntary and forced exercise on plaque deposition, hippocampal volume, and behavior in the Tg2576mouse model of Alzheimer's disease. Neurobiol. Dis. 35, 426–432.##Jankowsky, J.L., Xu, G., Fromholt, D., Gonzales, V., Borchelt, D.R. 2003. Environmental enrichment exacerbates amyloid plaque formation in a transgenic mouse model of Alzheimer disease. J. Neuropathol. Exp. Neurol. 62:1220–1227.##Zagaar, Munder, et al.2012. "The beneficial effects of regular exercise on cognition in REM sleep deprivation: behavioral, electrophysiological and molecular evidence." Neurobiology of disease 45.3: 1153-1162.##Doost Mohammadpour, J., Hosseinmardi, N., Janahmadi, M., et al. 2015. Non-selective NSAIDs improve the amyloid-β-mediated suppression of memory and synaptic plasticity. Pharmacology, Biochemistry and Behavior 132: 33–41.##Yaghoubi, Ali, Sahab-Joo, Marzieh; Fallah Mohammadi, Zia; Hedayati, Mahdi; Hajizadeh Moghadam, Akbar. The effect of continuous training intensity on the level of amyloid beta-42-1 hippocampus of alzheimerized mice with homocysteine injection. Journal of Arak University of Medical Sciences. 18 (11): 83-93. (Persian).##Nunan J, Shearman MS, Checler F, et al. 2001. The Cterminal fragment of the Alzheimer’s disease amyloid protein precursor is degraded by a proteasome-dependent mechanism distinct from γ-secretase. Eur J Biochem; 268(20): 5329 - 5336.##Cho JY, Um HS, Kang EB, et al. 2010. The combination of exercise training and α-lipoic acid treatment has therapeutic effects on the pathogenic phenotypes of Alzheimer's disease in NSE/APPsw-transgenic mice. Int J Mol Med; 25(3): 337-34.##Herring A, Yasin H, Ambrée O, et al. 2008. Environmental Enrichment Counteracts Alzheimer’s Neurovascular Dysfunction in TgCRND8 Mice. Brain Pathology 18:32-39.##Liu Q, Zhang J, Tran H, Verbeek MM, Reiss K, Estus S, Bu G. LRP1 shedding in human brain: roles of ADAM10 and ADAM17. Mol Neurodegener. 2009; 4:17.##Falah Mohammadi, Zia; Ebrahimzadeh, Mojtaba; Hajizadeh Moghadam, Akbar. 1392. The effect of 6 weeks of optional running on the levels of beta-amyloid beta (Aβ1-42) brain of alloxan-induced diabetic rats. Zahedan Journal of Research in Medical Sciences. 23-29. (Persian).##
)